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		<title><![CDATA[discussion boards — nc866 Dual Role in Cholangiocarcinoma]]></title>
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		<description><![CDATA[The most recent posts in nc866 Dual Role in Cholangiocarcinoma.]]></description>
		<lastBuildDate>Wed, 28 Nov 2012 21:27:03 +0000</lastBuildDate>
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			<title><![CDATA[nc866 Dual Role in Cholangiocarcinoma]]></title>
			<link>http://www.cholangiocarcinoma.org/punbb/viewtopic.php?pid=68647#p68647</link>
			<description><![CDATA[<p>Hi, everyone,</p><p>Consistently, artificial suppression of nc886 in cholangiocyte cells activates the canonical PKR/eIF2α cell death pathway, suggesting a potential significance of the nc886 suppression and the consequent PKR activation in eliminating <span class="bbu">pre-malignant cells during tumorigenesis.but...</span></p><p><a href="http://www.ncbi.nlm.nih.gov/pubmed/22926522">http://www.ncbi.nlm.nih.gov/pubmed/22926522</a></p><p>God bless.</p>]]></description>
			<author><![CDATA[null@example.com (PCL1029)]]></author>
			<pubDate>Wed, 28 Nov 2012 21:27:03 +0000</pubDate>
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